Energy Stack

Energy Stack includes:

• 1 x SS-31 50mg vial

• 1 x NAD+ 1000mg vial

• 1 x MOTS-C 40mg vial

Bacteriostatic Water is also included.

SS-31

SS-31, also known as Elamipretide, Bendavia, or MTP-131, is a synthetic peptide that targets and improves mitochondrial function. Mitochondria generate ATP (the body’s primary energy molecule), regulate cellular homeostasis, and impact nearly all physiological systems. SS-31 is a mitochondrial-targeted antioxidant with extensive therapeutic potential: mitigating oxidative stress, suppressing inflammation, maintaining mitochondrial health, and preventing cellular apoptosis.

It has several advantages over natural antioxidants and plays a key role in treating mitochondrial dysfunction-related disorders.

NAD+

NAD+ is a coenzyme that is involved in a broad number of biological functions. Starting around the age of 30, naturally occurring levels of NAD+ begin to decline, and continue to diminish as time passes. Because it is present in every living cell and aids in so many vital processes— regulating circadian rhythm, DNA repair functions, and immune system health—the decline in NAD+ levels can be linked to a number of health problems. These problems can include various cancers, neurodegenerative diseases like Alzheimer’s and Parkinson’s, and metabolic disorders.

MOTS-C

MOTS-C (Mitochondrial open reading frame of the 12S rRNA-c) is a peptide encoded in mitochondrial DNA. It improves glucose metabolism in skeletal muscle, which implies a number of benefits for those with diabetes, obesity, or who are over 30 years old. It is upregulated in response to exercise, and is considered an exercise mimetic.An exercise mimetic is a drug or compound that mimics the beneficial physiological effects of physical exercise on the body, essentially providing some of the health benefits of working out without the need for actual physical activity. It aims to activate similar cellular pathways triggered by obesity, diabetes, or muscle-wasting by simulating exercise-induced adaptations in the body. This provides a viable alternative to physical exercise in populations who are unable to exercise due to any number of limitations. Also, naturally occurring levels of MOTS-C diminish substantially throughout the aging process.Dysregulated MOTS-C levels have been associated with defective insulin secretion, insulin resistance, or both. This results in dysregulated mitochondrial activities such as mitochondrial fission and fusion, mitochondrial oxidative stress, and decreased NAD+ levels in the system. MOTS-C interacts with and regulates many nuclear genome-encoded proteins responsible for regulating metabolic adaptability and age-related diseases.